ライフサイエンスコーパス: stria vascularis

1 , forming the intermediate cell layer of the stria vascularis.

2 ner hair cells or a reduced thickness of the stria vascularis.

3 stemic circulation and the fluids inside the stria vascularis.

4 e progressive appearance of large lesions in stria vascularis.

5 articular in the cochlear organ of Corti and stria vascularis.

6 nd structurally damaged blood vessels in the stria vascularis.

7 at Kir4.1 confers the outflow of K(+) in the stria vascularis.

8 y regions between the sensory tissue and the stria vascularis.

9 melanocyte-derived intermediate cells in the stria vascularis.

10 lesions in the cochlea were found within the stria vascularis, a barrier epithelium containing the pr

11 In the previously reported S1pr2(-/-) mice, stria vascularis abnormalities, organ of Corti degenerat

12 ts principally from degeneration of cochlear stria vascularis and decline of the endocochlear potenti

13 ortance of basal cell tight junctions in the stria vascularis and directly verify the two-cell hypoth

14 Both marginal cells of the stria vascularis and hair cells express Trpml3 mRNA.

15 artery, which supplies blood directly to the stria vascularis and protects its capillary bed from hig

16 tissues, increased E2F1 and apoptosis in the stria vascularis and spiral ganglion neurons of the inne

17 Ngb was not present in the stria vascularis and the inner and outer hair cells.

18 tribution of nonsensory cell networks in the stria vascularis and the sensory region toward the matur

19 ing was restricted to the basal cells in the stria vascularis and was also detectable in the spiral g

20 an of Corti, in the basal cell region of the stria vascularis, and in type 1 fibrocytes of the spiral

21 We found that hair cells, marginal cells of stria vascularis, and other cells lining the cochlear an

22 was observed to leak from capillaries of the stria vascularis, and pericytes lost their tight associa

23 barrier properties of tight junctions of the stria vascularis appeared intact in a biotin tracer assa

24 in the human cochlea, and they point to the stria vascularis as an important therapeutic target for

25 licated apoptosis in the spiral ganglion and stria vascularis because of mitochondrial reactive oxyge

26 ments (organ of Corti, afferent neurons, and stria vascularis) can degenerate independently, and exer

27 paratus of renal glomerular podocytes and in stria vascularis cells of the inner ear, consistent with

28 high throughput of K(+) across cells of the stria vascularis, conferred partly by the activity of Ki

29 Gelatin zymography of extracts from the stria vascularis confirmed these findings.

30 berrations in outer and inner hair cells and stria vascularis defects, leading to deafness in the var

31 hepatocyte growth factor (HGF) signaling in stria vascularis development for the first time and that

32 ochlear potential, indicative of significant stria vascularis dysfunction, but without obvious signs

33 Aberrant splicing of Fgfr2 blocked stria vascularis formation due to erroneous ligand usage

34 he relation of this developmental process to stria vascularis function is currently unknown.

35 n of transcripts in the WT versus Nr3b2(-/-) stria vascularis has identified a set of genes that is l

36 The stria vascularis, however, does not sustain permanent da

37 le's loop and for potassium secretion by the stria vascularis in the inner ear.

38 n proteins in the endothelial barrier of the stria vascularis (intrastrial fluid-blood barrier) throu

39 The stria vascularis is a nonsensory structure that is essen

40 Vascular disturbance within the stria vascularis is a potential mechanism that leads to

41 embranes in the cochlea, suggesting that the stria vascularis is the primary site of cochlear pathoge

42 Explants of stria vascularis, 'mini-chips', are selectively cultured

43 ss structures of the endolymphatic space and stria vascularis observed at the light microscope level

44 acrophage-like melanocytes (PVM/Ms) from the stria vascularis of mice aged between P10 and P15 (P, po

45 anently impaired expression of KCNJ10 in the stria vascularis of Pit1(dw) mice, which likely contribu

46 rporation of the melanocytes into the future stria vascularis of the cochlear duct requires c-MET sig

47 owth factor (Hgf) is expressed in the future stria vascularis of the cochlear epithelium.

48 ll barriers and the capillary bed within the stria vascularis of the S1P(2) receptor-null mice showed

49 y outer hair cells (OHCs), inner hair cells, stria vascularis, spiral ganglia, and surrounding nerves

50 tivity to 8-isoprostane was increased in the stria vascularis, spiral ganglion cells and the organ of

51 vity delineated blood vessels located in the stria vascularis, spiral ligament, sub-basilar region, s

52 ing that the intermediate cells (ICs) of the stria vascularis (StV) express outward K+ current that r

53 The stria vascularis (StV) of the cochlear duct is the stati

54 tin accumulation is consistently high in the stria vascularis, the region of the cochlea that maintai

55 used a decrease in VEGF expression levels in stria vascularis vessels.

56 evealed that pericytes on capillaries of the stria vascularis were closely associated with the endoth

57 permeability of a biotin-based tracer in the stria vascularis were unaltered.

58 hin the organ of Corti, spiral ganglion, and stria vascularis, which are known targets of cisplatin o

59 ced by 4 and 8 weeks old in mutants, and the stria vascularis, which generates the EP, showed degener

60 The primary lesion was localized to the stria vascularis, which houses the main vasculature of t