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1 t most rVRG inspiratory premotor neurons are enkephalinergic.
2 r voluntary wheel running is associated with enkephalinergic activity in the ventral striatum is not
3 ed overlap exists between the populations of enkephalinergic and serotonergic neuronal afferents to t
4 present study was undertaken to determine if enkephalinergic compounds likewise have the ability to n
6 he thoracolumbar spinal cord receive a dense enkephalinergic (ENK) innervation from supraspinal sourc
10 ortex exerts a differential influence on the enkephalinergic (indirect pathway) as opposed to the sub
15 he hypothesis that under in vivo conditions, enkephalinergic nerves decrease and neurotensinergic ner
16 s located presynaptically on striatopallidal enkephalinergic neurons and by delta opioid receptors lo
17 ection neuron types by different mechanisms, enkephalinergic neurons by the trophic benefit of BDNF,
18 The densely labeled clusters of Fos-labeled enkephalinergic neurons occurred within larger zones con
19 The present study provides a complete map of enkephalinergic neurons within the forebrain and midbrai
24 come the inhibition to influence selectively enkephalinergic projection neurons in the activated matr
26 Silencing or activation of the dual GABA/enkephalinergic RVM neurons in adult animals substantial
28 iatum, partly rescued a preferential loss of enkephalinergic striatal neurons, and enhanced substance
29 sal pallidal neurons that receive input from enkephalinergic striatal neurons, as is true of STN.
30 ived neurotrophic factor-TrkB signaling from enkephalinergic striatal targets unexpectedly leads to s
31 ain-derived neurotrophic factor signaling in enkephalinergic striatopallidal neurons is poorly unders
32 suggest that enhanced responsiveness of the enkephalinergic system to alcohol is associated with, an
33 rs in the dorsal and ventral striatopallidal enkephalinergic system using fluorescence immunohistoche
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