ライフサイエンスコーパス: Fenton reaction

1 ion of free iron necessary for the genotoxic Fenton reaction.

2 tely cleaves DNA via a trace metal-dependent Fenton reaction.

3 ctive at inhibiting this classic O2-.-driven Fenton reaction.

4 , whose killing is amplified by iron via the Fenton reaction.

5 ultimately leading to cell apoptosis via the Fenton reaction.

6 tion and the degradation of H2O2 through the Fenton reaction.

7 age induced by hydroxyl radical generated in Fenton reaction.

8 e to attack by HO* radicals generated by the Fenton reaction.

9 of highly reactive hydroxyl radicals via the Fenton reaction.

10 ely oxidized by the radical generated by the Fenton reaction.

11 ecies, such as the hydroxyl radical, via the Fenton reaction.

12 cating that the DNA damage was caused by the Fenton reaction.

13 at H(2)O(2) rapidly oxidizes this metal in a Fenton reaction.

14 the biologically relevant superoxide-driven Fenton reaction.

15 me iron enzymes and in the first step of the Fenton reaction.

16 s against hydroxyl radicals generated by the Fenton reaction.

17 ial catalase and suppression of the damaging Fenton reaction.

18 s most likely not a major target for ROS via Fenton reaction.

19 ting hydroxyl-radical production through the Fenton reaction.

20 iron-sulfur clusters, and stimulation of the Fenton reaction.

21 y sensitive to nicking via the Fe2+-mediated Fenton reaction.

22 ion of free cysteine that fuels the damaging Fenton reaction.

23 ies produced by gamma-radiolysis of water or Fenton reaction.

24 d by hydroxyl radical generated from UHP via Fenton reaction.

25 ng the generation of hydroxyl radical by the Fenton reaction.

26 , the hydroxyl radical (*OH) produced by the Fenton reaction.

27 lled cells by damaging their DNA through the Fenton reaction.

28 mechanism to avoid the toxic effects of the Fenton reaction.

29 f free radical generation by formate via the Fenton reaction.

30 ng ferrous (Fe2+) form which can promote the Fenton reaction.

31 active and damaging hydroxyl radical via the Fenton reaction.

32 ociated with DNA in vivo, presumably via the Fenton reaction.

33 tion was OH., generated by a Cu(I)-catalyzed Fenton reaction.

34 sed by ionizing radiation and that caused by Fenton reactions.

35 3,4-DHPEA-EDA was oxidised by enzymatic and Fenton reactions.

36 uced injury to that thought to be induced by Fenton reactions.

37 iptional regulation, enzyme degradation, the Fenton reaction and damage caused by *OH, oxidation of b

38 supports our hypothesis that the synergy of Fenton reaction and manganese peroxidase might play an i

39 f hydroxyl radicals arises from an exogenous Fenton reaction and may stay either partially trapped on

40 The latter is mediated by Fenton reaction and prevents H2O2 accumulation.

41 lular iron levels; it thereby attenuates the Fenton reaction and the DNA damage that would otherwise

42 whereas O(2)(*-) (ONOO(-) formation), (*)OH (Fenton reaction), and compound III are unlikely to contr

43 OS) formed from gamma-radiolysis of water or Fenton reaction, and it can abstract one hydrogen atom f

44 In this work, we develop the electro-Fenton reaction as a means to generate hydroxyl radicals

45 Analogue 1c inhibited the Fe(II)-catalyzed Fenton reaction at about the same concentrations as asco

46 (50) 53 muM), inhibiting the Cu(I)-catalyzed Fenton reaction at lower concentrations than GSH, ascorb

47 Compound 7A inhibited Fenton reaction better than EDTA, IC50 of 37 vs 54 muM,

48 Supporting the role of killing via the Fenton reaction, binding of iron by Dps significantly mi

49 E. coli eliminates substrates of the Fenton reaction by assimilating Fe(2+) and biosynthesizi

50 l formation by eliminating substrates of the Fenton reaction, by assimilating ferrous iron (Fe(2+)) a

51 The microbially driven Fenton reaction completely degraded 1,4-dioxane (10 mM i

52 ins that are carbonylated in a receptor- and Fenton reaction-dependent manner, including annexin A1,

53 The Fenton reaction describes the reaction of Fe(II) with hy

54 This unexpected effect of pH on Fenton reaction efficiency could be due to the formation

55 ted ethanol in the presence of Fe(2+) by the Fenton reaction, establishing an acidic milieu.

56 roxide photolysis (H2O2 + hnu) and the photo-Fenton reaction (Fe (II) + H2O2 + hnu).

57 attenuate hydroxyl radical production by the Fenton reaction (Fe(2+) + H(2)O(2) --> Fe(3+) + OH(-) +

58 Fenton reactions (Fe(2+)/Fe(3+) plus H2O2) were used for

59 footprinting has been developed based on the Fenton reaction, Fe(II) + H2O2 --> Fe(III) + *OH + OH-.

60 ctive hydroxyl radical (.OH) [formed via the Fenton reaction (Fe2++H2O2+H+-->Fe3++H2O+.OH)], interfer

61 the generation of free radicals through the Fenton reaction, ferritin acts as an anti-oxidant.

62 sistance was not caused by inhibition of the Fenton reaction, for copper-supplemented cells exhibited

63 n binding capacity, oxidation of rRNA by the Fenton reaction formed 13 times more 8-hydroxyguanosine

64 In the Fenton reaction, free intracellular iron transfers elect

65 and II ionic species, and superoxide but not Fenton reaction generated hydroxyl radicals, are importa

66 important source of hydroxyl radical via the Fenton reaction in cloudwater.

67 tion of hydroxyl radical ((*)OH) through the Fenton reaction in mitochondria.

68 es generated in vivo and the key role of the Fenton reaction in this process may be important for und

69 sal that NADH was the reducing agent for the Fenton reaction in vivo.

70 The hydroxyl radical ions produced via the Fenton reaction inactivate GTF, a factor in the producti

71 ding not only synchrotron radiation but also Fenton reactions involving chelated iron, have become an

72 the damage spectrum of the dC family by the Fenton reaction is compared with that by ionizing radiat

73 In contrast to restriction enzymes the Fenton reaction is known to cleave DNA without nucleotid

74 The Fenton reaction is used to produce hydroxyl radicals for

75 e hydroxyl radical, which is produced in the Fenton reaction, is buffered by extracellular proteins,

76 Free iron, via the Fenton reaction, is known to exacerbate UV-induced and o

77 The rate constant of the Fenton reaction measured at physiological pH was much hi

78 DNA is damaged in vivo by the Fenton reaction mediated by Fe2+ and cellular reductants

79 d generation of free radical species, by the Fenton reaction, might contribute to the pathoetiology o

80 Nicking of duplex DNA by the iron-mediated Fenton reaction occurs preferentially at a limited numbe

81 ic) Fenton reactions, the microbially driven Fenton reaction operated at circumneutral pH and did not

82 by generating reactive oxygen species due to Fenton reaction or by substituting for other transition

83 ork, dG was oxidized by HO(*) via the Fe(II)-Fenton reaction or by X-ray radiolysis of water.

84 cted HLECs were exposed to oxidative stress (Fenton reaction) or HNE (30 microM) for 3 hours.

85 , it has been examined to what extent adding Fenton reaction promoting Fe impacted the toxicity of an

86 The microbially driven Fenton reaction provides the foundation for development

87 hat vitamin C, a compound known to drive the Fenton reaction, sterilizes cultures of drug-susceptible

88 etter than other antioxidants in a chemical (Fenton reaction) system and neuronal cultures.

89 so leads to the production of free radicals (Fenton reaction) that can attack and damage lipids, prot

90 The Fenton reaction, the oxidation of ferrous iron by hydrog

91 se iron-loaded enzymes are vulnerable to the Fenton reaction, the substitution of manganese may preve

92 comparison to conventional (purely abiotic) Fenton reactions, the microbially driven Fenton reaction

93 set of oxidative reactions in analogy to the Fenton reaction, thus widening the scope of electrochemi

94 NADH could drive the Fenton reaction to cause damage to the dC family in vitr

95 d synergistically enhances the efficiency of Fenton reaction to degrade pectin into 5.5 kDa within on

96 n of pentosidine and carboxymethyllysine via Fenton reaction (UHP-Fenton pathway).

97 Damage by iron-mediated Fenton reactions under aerobic or anaerobic conditions t

98 In the present study, a microbially driven Fenton reaction was designed to autocatalytically genera

99 rformance of BPA oxidation in an EDDS-driven Fenton reaction was found to be much higher at near neut

100 roxyl-radical formation, confirming that the Fenton reaction was responsible.

101 nd their ability to inhibit .OH formation in Fenton reactions was quantified by ESR measurements.

102 s of DMPO/.17OH and DMPO/.16OH formed in the Fenton reaction were 90% and 10%, respectively, reflecti

103 onols quercetin and kaempferol on iron-based Fenton reaction were documented.

104 by the presence of O2 during Fe(2+)-mediated Fenton reactions when H2O2 is in excess.

105 ould be involved in avoiding metal-dependent Fenton reactions when photooxidation causes disassembly

106 es in dental caries by taking advantage of a Fenton reaction which requires metal ions such as iron o

107 ve hydroxyl radical that is generated by the Fenton reaction with H2O2, might contribute to the sourc